TNIK as a Potential Target for Age-Slowing Therapeutics
The search for new mechanisms of action in cell metabolism is a continuous endeavor in academia and the pharmaceutical industry. While some areas of metabolism remain uncharted, they hold promise for potential interventions. Traf2- and Nck-interacting kinase (TNIK) has recently emerged as a key regulator with implications for cancer, metabolic disorders, and neuronal function. Its role in cellular senescence, nutrient sensing, inflammation, and intercellular communication suggest a possible link to aging-related pathology. Understanding TNIK dysregulation could provide insights into the aging process and its associated diseases.
Read more about TNIK and its potential impact on aging-related diseases in the full article.
